dementia

DARSHAN SAPKOTA An extended form of the protein aquaporin 4 (red) lines the edges of tiny blood vessels in the brain. Cell nuclei are visible in blue. Researchers at Washington University School of Medicine in St. Louis have found a new druggable pathway that enhances the amount of long aquaporin 4 near blood vessels and increases the clearance of waste from the brain. The findings potentially could lead to new therapies to prevent Alzheimer’s dementia.

A new approach to clearing toxic waste from the brain

Microglia (green) derived from human stem cells by members of the Kampmann lab. Image by Olivia Teter, Kampmann lab, UCSF

The opportunity for an entirely new type of therapeutic approach to treat Alzheimer’s and other forms of dementia

An artificial intelligence program that detects cognitive impairment accurately and efficiently from voice recordings

New genetic research shows a direct link between dementia and a lack of vitamin D

Could smart socks help millions living with dementia?

Could an essential amino acids supplement keep dementia at bay?

Identifying a likely cause of Alzheimer’s

Researchers have successfully and reproducibly created clinical-grade transplantable brain cells in the lab

Could apathy predict the onset of some forms of dementia many years before symptoms start?

Is a possible dementia vaccine closer?

A new way to treat or prevent Alzheimer’s disease?

A new approach using 2 old drugs show promise for specific types of stroke and dementia

Robot helpers for the joy and sorrow of dementia

Could readily available plant-based supplements offer protection against dementia?

Using ultrasound to delay the effects of dementia